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Here, we discuss the role of oxidative/nitrative/nitrosative stress in ASD pathogenesis

Acylcarnitines that accumulate through disordered carnitine metabolism directly inhibit the surface activity of pulmonary surfactants and are considered risk factors for increased lung injury due to a second hit, such as respiratory tract infection, sepsis, or inhalation of toxins, in patients with dysregulated FAO [304, 305]

It shuttles fatty acids across the mitochondrial membrane for beta-oxidation, helping convert fat into ATP while also buffering toxic acyl groups

Acylcarnitine metabolomic profiles inform clinically-defined major depressive phenotypes